The human body can be envisioned as a grand symphony orchestra, where individual organs act as musicians, and the conductor is the brain. As long as the orchestra plays in harmony under the conductor’s baton, the individual remains healthy. However, aging introduces a series of disruptions—some musicians begin to play out of tune, and at times, the conductor starts losing control over the ensemble. The most common disorder associated with aging is the incorrect guidance of the orchestra by the conductor (the brain), leading to errors among the musicians (organs). Consequently, the entire symphony loses synchronization, and ultimately, the concert comes to an end—representing death.
Following this metaphorical model of aging, we recognize that the transmission of information from the conductor (brain) to the distant musicians (organs) occurs at an exceptionally high speed. As previously analyzed, neuronal communication operates at speeds ranging from tens to hundreds of meters per second, depending on the type of nerve fiber. This rapid transmission can only be achieved through a combination of electrical and electromagnetic signals. Such mechanisms inherently involve processes occurring at the subatomic level, placing them within the domain of electronic biology (https://electronicbiology.org/).
Understanding the physical foundation of these communication processes opens new possibilities for influencing aging. By correcting errors within the symphony orchestra (the body), whether they originate from the conductor (the brain) or the musicians (the organs), we may be able to mitigate or even reverse aspects of aging. This perspective shifts the focus of aging research from purely biochemical interventions to an electronic and informational approach, where the synchronization of signals and the maintenance of coherent bioelectronic interactions become essential in sustaining health and prolonging life.
Through this lens, aging is not merely a gradual accumulation of cellular damage but a progressive desynchronization of biological information networks. Suppose we can refine our understanding of these fundamental electronic interactions. In that case, we may unlock new strategies to restore harmony to the orchestra of life-extending not just lifespan, but healthspan as well.